Acknowledgements
Trevor Bedford and his lab -- terrific collaboration since 2014
James Hadfield, Emma Hodcroft and Tom Sibley have led the recent development
Data we analyze are contributed by scientists from all over the world
Data are shared and curated by GISAID
SARS-CoV-2 and its relatives
SARS-CoV-2 is in the same family as SARS-CoV-1, MERS and two seasonal coronavirus
The latter cause mild disease, the former severe
SARS-CoV-2 spreads more easily than SARS-CoV-1 or MERS
Relatives are found in many different animals
Closest known relative is a virus isolated from bats (RaTG13, approx 96% identical)
Virus genomes change rapidly through time
A/Brisbane/100/2014
GGATAATTCTATTAACCATGAAGACTATC ATTGCTTT...
A/Brisbane/1000/2015
GGATAATTCTATTAACCATGAAGACTATT ATTGCTTT...
A/Brisbane/1/2017
GGATAATTCTATTAACCATGAAGACTATC ATTGCTTT...
... tens of thousands of sequences...
Genome sequences record the spread of pathogens
in SARS-CoV-2 a new mutation accumulates about every 2 weeks
images by Trevor Bedford
Early lessons from SARS-CoV-2 genomes (mid Jan)
The outbreak originated from one source
→ not repeated zoonoses from a diverse reservoir.
The common ancestor of all samples was Nov - early Dec 2019.
Family clusters showed up as identical genomes (expected)
A second clade emerged that will continue to spread
→ the closest to "real-time" we have experienced so far...
Figure by James Hadfield/Emma Hodcroft
Available data on June 4
The virus has reached most places where humans live
nextstrain.org/ncov
Available data on June 4
Different clades (virus variants) dominate in different parts of the US
nextstrain.org/ncov
Available data on June 4
The outbreak in New York was likely seeded by introductions from Europe
nextstrain.org/ncov
Current take-home from SARS-CoV-2 genomes
SARS-CoV-2 was disseminated widely before travel restrictions
→ Border closures had little effect (maybe if earlier)
The virus typically spread locally before it was noticed
(while testing focussed on people with travel history to hotspots)
Temporal resolution by mutation is about 4 weeks
→ too low to resolve direct transmissions or directionality
→ sufficient to connect outbreaks and identify clusters
Currently no solid evidence for mutations with functional significance
see Sergei Pond's analysis of signals of positive selection
The history of nextstrain -- KITP in summer 2014
Trevor Bedford, FHCRC Seattle
Conspiracy theories and sensationalist research
WRONG:
LIKELY WRONG:
Prevalence is high, the mortality is low
False positive rates are not accounted for correctly, unrepresentative study populations
Some strains are more severe than others
confounders not accounted for
Some strains are adapted to different parts of the world
confounders not accounted for
many more....
Colorful trees are easily misinterpreted...
Low genetic diversity combined with very biased sampling → no directionality can be inferred
nextstrain.org/ncov
Acknowledgements
Trevor Bedford and his lab -- terrific collaborations since 2014
James Hadfield, Emma Hodcroft, and Tom Sibley have led the recent development
People at the frontlines: health care staff and everybody else who keeps things going...
Data we analyze are contributed by scientists all over the world
Data are shared and curated by GISAID
Seasonal variation in infectivity
Original title mid February...
Published two weeks later...
Potential seasonal drivers
humidity (e.g. dry air increases time before droplets fall down)
temperature
UV light
behavior (e.g. time spent indoors, ventilation, heating)
Exact nature of forcing not relevant for this talk...
Human corona viruses have pronounced seasonal prevalence (Sweden)
Neher et al
SIR model with seasonal forcing
$$\beta(t) = \beta_0\left(1+\epsilon\cos(2\pi (t-\theta))\right)$$
People import new infections at rate $\nu$
Neher et al
Strong seasonality can be generated through strong forcing or resonance
Yellow: good fit -- Blue: poor fit
Neher et al
Seasonal forcing and the SARS-CoV-2 pandemic
Counter-factual scenario without strong social distancing!
Seasonal forcing and the SARS-CoV-2 pandemic
Potential transition to an endemic seasonal virus